That was really interesting and useful information. But I wanted to ask you about what Dom said at time (52min). What does he mean,when he says 1 mml increase is about 10% increase of substrate to the brain? Is that (same glucose amount in brain) + (typical ketone amount in brain)+ and 10% more? Or does it substitute glucose? And if it is so, does the brain use that energy or does it just stay around the brain without being used? I know we can’t know for sure, but it would be helpful if you said what you think. Thanks in advance!
In nondiabetics, ketosis (also called nutritional ketosis) is regulated and controlled in the body so that ketone levels never reach the harmful levels associated with diabetic ketoacidosis. Ketoacidosis is an acute, life-threatening condition that occurs in severely uncontrolled diabetes (mainly type 1) when ketones rise to massive, supranormal levels.
Now I though about taking keto supplement to boost my ketosis (I’m also looking to boost my overall electrolyte and vitamin/mineral). I’m unsure what product to take. Should I take keto os or ketocana (or else)? Should I take it in the morning while I’m empty stomack for the next 6 hours (wake up at 5) with my mg cap so I get a boost in electrolyte and keto in the morning? Should I take more? Depending on the set, I might not have to take mg cap anymore since they all include some in it.

This answer made me sad. Having lived in New York all of my life, I from a young age saw loved ones fall victim to spontaneous gluten intolerance and restaurants run to their rescue with accommodating menus.  This is why I get annoyed. What’s a dietary prescription for someone who wants to go to a dinner party and be able to eat a meal that was prepared for them? Keto is cultish not for its members’ radical commitment to a dietary restriction but for their lack of full participation in the outside world.

In sheep, ketosis, evidenced by hyperketonemia with beta-hydroxybutyrate in blood over 0.7 mmol/L, occurs in pregnancy toxemia.[78][79] This may develop in late pregnancy in ewes bearing multiple fetuses,[78][79] and is associated with the considerable glucose demands of the conceptuses.[80][81] In ruminants, because most glucose in the digestive tract is metabolized by rumen organisms, glucose must be supplied by gluconeogenesis,[82] for which propionate (produced by rumen bacteria and absorbed across the rumen wall) is normally the principal substrate in sheep, with other gluconeogenic substrates increasing in importance when glucose demand is high or propionate is limited.[83][84] Pregnancy toxemia is most likely to occur in late pregnancy because most fetal growth (and hence most glucose demand) occurs in the final weeks of gestation; it may be triggered by insufficient feed energy intake (anorexia due to weather conditions, stress or other causes),[79] necessitating reliance on hydrolysis of stored triglyceride, with the glycerol moiety being used in gluconeogenesis and the fatty acid moieties being subject to oxidation, producing ketone bodies.[78] Among ewes with pregnancy toxemia, beta-hydroxybutyrate in blood tends to be higher in those that die than in survivors.[85] Prompt recovery may occur with natural parturition, Caesarean section or induced abortion. Prevention (through appropriate feeding and other management) is more effective than treatment of advanced stages of ovine ketosis.[86]
They mimic ketosis, to answer your question in short. Best to get fat adapted and use ketones to get the best of both worlds and to enhance fat burning abilities. They will allow you to use glucose and fatty acids simultaneously, yes. Anyways, I'd be happy to help you via a personal one-on-one consult. Just go to and then choose a 20 or 60 minute consult, whichever you'd prefer. I can schedule ASAP after you get that.

For any long 90+ minute workouts or competitions for which glycogen depletion is a potential issue, use Glycofuse, but use half of the recommended serving of it, and add one scoop of Catalyte electrolytes, one scoop of Aminos, and one serving of medium chain triglycerides in the form of Brain Octane, KetoCaNa or KETO//OS (pick your poison, it’s up to you).
Can’t get enough of avocados? You now have a great excuse to eat more of them. A ½-cup serving of the creamy fruit has almost 12 g of fat and only 2.6 g of net carbs. Avocados are also low in calories (about 138 for the same serving), making them an ideal snack in between meals. One serving also has about 6.4 g of dietary fiber (25.6 percent daily value, or DV), 404 milligrams (mg) of potassium (8.6 percent DV), and only 2.8 g of sugar. Try topping your salad with cubed avocado for a keto-friendly lunch.

The thing is, for keto to work, it can’t just be treated like any other diet, which is really confusing considering all the pundits touting the “keto diet.” Eating a balanced diet in daylight hours and night capping with a “keto bar” from Whole Foods is not going to put you in a state of ketosis, nor is eating keto religiously and bingeing the family sized bag of Doritos twice a week. Putting the word “diet” beside “keto” is a bit misleading because it downplays the outrageous restrictions required to put your body in an unnatural fat-burning state. In order to accomplish the “ketosis” most diet plans talk about, you need to radically change your lifestyle -- and, unlike switching to a balanced diet of whole foods, you must be fairly religious about carb restriction in order to reap the benefits.